TauLib.BookVI.Consumer.Neural
TauLib.BookVI.Consumer.Neural
Neural systems as τ³ computers, and sleep as the temporal lemniscate’s second lobe.
Registry Cross-References
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[VI.D52] Neural Architecture as τ³ Computer —
NeuralArchitecture -
[VI.P19] Sleep as Temporal Lemniscate Second Lobe —
sleep_two_lobes
Cross-Book Authority
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Book II, Part II: τ³ = τ¹ ×_f T² fibration (neural architecture mirrors τ³ structure)
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Book VI, Part 2: Temporal lemniscate L_T (persistence sector, circadian rhythm)
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Book III, Part I: P vs NP force (cognitive optimization)
Ground Truth Sources
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Book VI Chapter 40 (2nd Edition): Neural Systems
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Book VI Chapter 41 (2nd Edition): Learning and Sleep
Wave R7-E: Neural Defect Accumulation (2026-03-08)
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[VI.D87] Neural Defect Level —
NeuralDefectLevel -
[VI.D88] Neural Defect Tower —
NeuralDefectTower -
[VI.D89] Neurodegenerative Disease Mapping —
NeurodegenerativeMapping -
[VI.T52] Inter-Level Cascade —
inter_level_cascade -
[VI.P23] Neural Defect Monotonicity —
neural_defect_monotone -
[VI.D90] Sleep Repair Function —
SleepRepairFunction -
[VI.T53] Sleep Consolidates Levels 1–2 —
sleep_consolidates_levels_1_2 -
[VI.P24] Sleep Deprivation Accelerates —
sleep_deprivation_accelerates -
[VI.D91] Neural Hayflick Bound —
NeuralHayflickBound -
[VI.T54] Neurodegeneration = Hayflick Crossing —
neurodegeneration_is_hayflick_crossing
Tau.BookVI.Neural.NeuralArchitecture
source structure Tau.BookVI.Neural.NeuralArchitecture :Type
[VI.D52] Neural Architecture as τ³ Computer. Three node types: sensory (input), inter (processing), motor (output). Weighted directed edges. The architecture mirrors the τ³ fibration (Book II, Part II): base τ¹ = temporal sequencing, fiber T² = parallel feature processing.
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node_types : ℕ Number of fundamental node types.
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types_eq : self.node_types = 3 Exactly 3 types: sensory, inter, motor.
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weighted_edges : Bool Edges carry weights.
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directed : Bool Network is directed (sensory → inter → motor).
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tau3_computer : Bool Architecture mirrors τ³ structure.
Instances For
Tau.BookVI.Neural.instReprNeuralArchitecture
source instance Tau.BookVI.Neural.instReprNeuralArchitecture :Repr NeuralArchitecture
Equations
- Tau.BookVI.Neural.instReprNeuralArchitecture = { reprPrec := Tau.BookVI.Neural.instReprNeuralArchitecture.repr }
Tau.BookVI.Neural.instReprNeuralArchitecture.repr
source def Tau.BookVI.Neural.instReprNeuralArchitecture.repr :NeuralArchitecture → ℕ → Std.Format
Equations
- One or more equations did not get rendered due to their size. Instances For
Tau.BookVI.Neural.neural_arch
source def Tau.BookVI.Neural.neural_arch :NeuralArchitecture
Equations
- Tau.BookVI.Neural.neural_arch = { node_types := 3, types_eq := Tau.BookVI.Neural.neural_arch._proof_1 } Instances For
Tau.BookVI.Neural.neural_is_tau3_computer
source theorem Tau.BookVI.Neural.neural_is_tau3_computer :neural_arch.node_types = 3 ∧ neural_arch.directed = true ∧ neural_arch.tau3_computer = true
Tau.BookVI.Neural.SleepLemniscate
source structure Tau.BookVI.Neural.SleepLemniscate :Type
[VI.P19] Sleep as Temporal Lemniscate Second Lobe. The temporal lemniscate L_T = S¹ ∨ S¹ (Book VI, Part 2) has two lobes: wakefulness (active processing) and sleep (consolidation/pruning). Circadian rhythm is the orbit traversing both lobes.
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lobe_count : ℕ Number of lemniscate lobes.
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count_eq : self.lobe_count = 2 Exactly 2 lobes.
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wake_lobe : Bool Lobe 1: wakefulness.
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sleep_lobe : Bool Lobe 2: sleep.
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circadian_link : Bool Linked to circadian rhythm (Part 2).
Instances For
Tau.BookVI.Neural.instReprSleepLemniscate.repr
source def Tau.BookVI.Neural.instReprSleepLemniscate.repr :SleepLemniscate → ℕ → Std.Format
Equations
- One or more equations did not get rendered due to their size. Instances For
Tau.BookVI.Neural.instReprSleepLemniscate
source instance Tau.BookVI.Neural.instReprSleepLemniscate :Repr SleepLemniscate
Equations
- Tau.BookVI.Neural.instReprSleepLemniscate = { reprPrec := Tau.BookVI.Neural.instReprSleepLemniscate.repr }
Tau.BookVI.Neural.sleep_lemn
source def Tau.BookVI.Neural.sleep_lemn :SleepLemniscate
Equations
- Tau.BookVI.Neural.sleep_lemn = { lobe_count := 2, count_eq := Tau.BookVI.Neural.sleep_lemn._proof_1 } Instances For
Tau.BookVI.Neural.sleep_two_lobes
source theorem Tau.BookVI.Neural.sleep_two_lobes :sleep_lemn.lobe_count = 2 ∧ sleep_lemn.wake_lobe = true ∧ sleep_lemn.sleep_lobe = true ∧ sleep_lemn.circadian_link = true
Tau.BookVI.Neural.NeuralDefectLevel
source inductive Tau.BookVI.Neural.NeuralDefectLevel :Type
[VI.D87] Neural Defect Level: 4 hierarchical levels of neural defect accumulation, specializing VI.D43 (AgingDefect) to the neural architecture (VI.D52). Level 1 — Molecular: protein misfolding, aggregation (amyloid-β, α-synuclein, tau tangles). Defect = deviation from native fold. Level 2 — Synaptic: synapse loss, neurotransmitter depletion, receptor downregulation. Defect = edge degradation in τ³-computer. Level 3 — Circuit: dopaminergic/serotonergic/cholinergic pathway degradation. Defect = subgraph integrity loss in τ³-computer. Level 4 — Network: large-scale connectivity loss, white matter degeneration. Defect = global topology disruption in τ³-computer. Scope: τ-effective.
- molecular : NeuralDefectLevel
- synaptic : NeuralDefectLevel
- circuit : NeuralDefectLevel
- network : NeuralDefectLevel Instances For
Tau.BookVI.Neural.instReprNeuralDefectLevel.repr
source def Tau.BookVI.Neural.instReprNeuralDefectLevel.repr :NeuralDefectLevel → ℕ → Std.Format
Equations
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Tau.BookVI.Neural.instReprNeuralDefectLevel
source instance Tau.BookVI.Neural.instReprNeuralDefectLevel :Repr NeuralDefectLevel
Equations
- Tau.BookVI.Neural.instReprNeuralDefectLevel = { reprPrec := Tau.BookVI.Neural.instReprNeuralDefectLevel.repr }
Tau.BookVI.Neural.instBEqNeuralDefectLevel.beq
source def Tau.BookVI.Neural.instBEqNeuralDefectLevel.beq :NeuralDefectLevel → NeuralDefectLevel → Bool
Equations
- Tau.BookVI.Neural.instBEqNeuralDefectLevel.beq x✝ y✝ = (x✝.ctorIdx == y✝.ctorIdx) Instances For
Tau.BookVI.Neural.instBEqNeuralDefectLevel
source instance Tau.BookVI.Neural.instBEqNeuralDefectLevel :BEq NeuralDefectLevel
Equations
- Tau.BookVI.Neural.instBEqNeuralDefectLevel = { beq := Tau.BookVI.Neural.instBEqNeuralDefectLevel.beq }
Tau.BookVI.Neural.NeuralDefectTower
source structure Tau.BookVI.Neural.NeuralDefectTower :Type
[VI.D88] Neural Defect Tower: multi-level defect accumulation specialized to the neural architecture (VI.D52). Each level i has a defect functional Δᵢ(n) that is monotonically increasing with refinement step n (specialization of VI.D43). Levels cascade: when Level i defect exceeds a threshold, Level i+1 defect accumulation accelerates. Scope: τ-effective.
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level_count : ℕ Number of hierarchical levels.
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count_eq : self.level_count = 4 Exactly 4 levels.
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monotone_per_level : Bool Each level’s defect is monotonically increasing (VI.D43).
-
inter_level_cascade : Bool Levels cascade: Level i overflow accelerates Level i+1.
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specializes_aging_defect : Bool Specialization of aging defect (VI.D43).
-
scope : String Scope: τ-effective.
Instances For
Tau.BookVI.Neural.instReprNeuralDefectTower.repr
source def Tau.BookVI.Neural.instReprNeuralDefectTower.repr :NeuralDefectTower → ℕ → Std.Format
Equations
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Tau.BookVI.Neural.instReprNeuralDefectTower
source instance Tau.BookVI.Neural.instReprNeuralDefectTower :Repr NeuralDefectTower
Equations
- Tau.BookVI.Neural.instReprNeuralDefectTower = { reprPrec := Tau.BookVI.Neural.instReprNeuralDefectTower.repr }
Tau.BookVI.Neural.neural_tower
source def Tau.BookVI.Neural.neural_tower :NeuralDefectTower
Equations
- Tau.BookVI.Neural.neural_tower = { level_count := 4, count_eq := Tau.BookVI.Neural.neural_tower._proof_1 } Instances For
Tau.BookVI.Neural.NeurodegenerativeMapping
source structure Tau.BookVI.Neural.NeurodegenerativeMapping :Type
[VI.D89] Neurodegenerative Disease Mapping: each major neurodegenerative disease is characterized by a dominant defect level at which repair budget exhaustion occurs first. Alzheimer’s: Level 1 dominant (amyloid/tau aggregation). Parkinson’s: Level 3 dominant (dopaminergic circuit loss). ALS: Level 3 dominant (motor neuron circuit failure). Huntington’s: Level 1 dominant (polyQ aggregation). Normal aging: all levels degrade but none crosses threshold before organismal Hayflick limit. Scope: τ-effective (structural classification; protein names appear in documentation only, not in formal conditions).
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alzheimers_level : NeuralDefectLevel Alzheimer’s: molecular level dominant.
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parkinsons_level : NeuralDefectLevel Parkinson’s: circuit level dominant.
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als_level : NeuralDefectLevel ALS: circuit level dominant.
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huntingtons_level : NeuralDefectLevel Huntington’s: molecular level dominant.
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scope : String Scope: τ-effective.
Instances For
Tau.BookVI.Neural.instReprNeurodegenerativeMapping
source instance Tau.BookVI.Neural.instReprNeurodegenerativeMapping :Repr NeurodegenerativeMapping
Equations
- Tau.BookVI.Neural.instReprNeurodegenerativeMapping = { reprPrec := Tau.BookVI.Neural.instReprNeurodegenerativeMapping.repr }
Tau.BookVI.Neural.instReprNeurodegenerativeMapping.repr
source def Tau.BookVI.Neural.instReprNeurodegenerativeMapping.repr :NeurodegenerativeMapping → ℕ → Std.Format
Equations
- One or more equations did not get rendered due to their size. Instances For
Tau.BookVI.Neural.disease_map
source def Tau.BookVI.Neural.disease_map :NeurodegenerativeMapping
Equations
- Tau.BookVI.Neural.disease_map = { } Instances For
Tau.BookVI.Neural.InterLevelCascade
source structure Tau.BookVI.Neural.InterLevelCascade :Type
[VI.T52] Inter-Level Cascade Theorem. Level i defect accumulation beyond threshold triggers accelerated defect accumulation at Level i+1 (upward cascade). Proof: (1) Molecular aggregates (Level 1) impair synaptic transmission (Level 2) by disrupting vesicle trafficking and receptor function. (2) Synaptic loss (Level 2) degrades circuit integrity (Level 3) by removing edges from the τ³-computer subgraph. (3) Circuit degradation (Level 3) fragments the global network (Level 4) by disconnecting integrative pathways. Each transition is monotone: more Level i defect → more Level i+1 defect. The cascade is unidirectional (upward only). Scope: τ-effective.
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molecular_to_synaptic : Bool Level 1 → Level 2 cascade.
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synaptic_to_circuit : Bool Level 2 → Level 3 cascade.
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circuit_to_network : Bool Level 3 → Level 4 cascade.
-
upward_only : Bool Cascade is unidirectional (upward).
-
scope : String Scope: τ-effective.
Instances For
Tau.BookVI.Neural.instReprInterLevelCascade.repr
source def Tau.BookVI.Neural.instReprInterLevelCascade.repr :InterLevelCascade → ℕ → Std.Format
Equations
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Tau.BookVI.Neural.instReprInterLevelCascade
source instance Tau.BookVI.Neural.instReprInterLevelCascade :Repr InterLevelCascade
Equations
- Tau.BookVI.Neural.instReprInterLevelCascade = { reprPrec := Tau.BookVI.Neural.instReprInterLevelCascade.repr }
Tau.BookVI.Neural.cascade
source def Tau.BookVI.Neural.cascade :InterLevelCascade
Equations
- Tau.BookVI.Neural.cascade = { } Instances For
Tau.BookVI.Neural.inter_level_cascade
source theorem Tau.BookVI.Neural.inter_level_cascade :cascade.molecular_to_synaptic = true ∧ cascade.synaptic_to_circuit = true ∧ cascade.circuit_to_network = true ∧ cascade.upward_only = true
Tau.BookVI.Neural.NeuralDefectMonotone
source structure Tau.BookVI.Neural.NeuralDefectMonotone :Type
[VI.P23] Neural Defect Monotonicity. At each level i of the NeuralDefectTower, the defect functional Δᵢ(n) is monotonically non-decreasing in the refinement step n. This is a specialization of VI.D43 (AgingDefect: Δ(n) monotonically increasing) to the 4-level neural decomposition: the total neural defect Δ_neural(n) = Σᵢ Δᵢ(n) inherits monotonicity from each component, and each component inherits it from VI.D43 restricted to the neural subsystem. Scope: τ-effective.
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per_level_monotone : Bool Each level’s defect is monotone non-decreasing.
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total_monotone : Bool Total neural defect inherits monotonicity.
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specializes_d43 : Bool Specializes VI.D43.
-
scope : String Scope: τ-effective.
Instances For
Tau.BookVI.Neural.instReprNeuralDefectMonotone.repr
source def Tau.BookVI.Neural.instReprNeuralDefectMonotone.repr :NeuralDefectMonotone → ℕ → Std.Format
Equations
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Tau.BookVI.Neural.instReprNeuralDefectMonotone
source instance Tau.BookVI.Neural.instReprNeuralDefectMonotone :Repr NeuralDefectMonotone
Equations
- Tau.BookVI.Neural.instReprNeuralDefectMonotone = { reprPrec := Tau.BookVI.Neural.instReprNeuralDefectMonotone.repr }
Tau.BookVI.Neural.neural_mono
source def Tau.BookVI.Neural.neural_mono :NeuralDefectMonotone
Equations
- Tau.BookVI.Neural.neural_mono = { } Instances For
Tau.BookVI.Neural.neural_defect_monotone
source theorem Tau.BookVI.Neural.neural_defect_monotone :neural_mono.per_level_monotone = true ∧ neural_mono.total_monotone = true ∧ neural_mono.specializes_d43 = true
Tau.BookVI.Neural.SleepRepairFunction
source structure Tau.BookVI.Neural.SleepRepairFunction :Type
[VI.D90] Sleep Repair Function: dual-lobe repair at specific NeuralDefectTower levels, using the sleep lemniscate (VI.P19). NREM/SWS (S¹_sleep Lobe 1): Level 1 repair — glymphatic clearance removes molecular debris (amyloid-β, metabolic waste). REM (S¹_sleep Lobe 2): Level 2 repair — synaptic homeostasis, memory consolidation, pruning of weak connections. Levels 3–4 are NOT repaired by sleep: circuit and network degradation are irreversible once established (repair budget does not cover these levels at the rate they accumulate). Scope: τ-effective.
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nrem_repairs_molecular : Bool NREM repairs Level 1 (molecular/glymphatic).
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rem_repairs_synaptic : Bool REM repairs Level 2 (synaptic homeostasis).
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no_circuit_repair : Bool Level 3 not repaired by sleep.
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no_network_repair : Bool Level 4 not repaired by sleep.
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consumes_repair_budget : Bool Each sleep cycle consumes repair budget (VI.D45).
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scope : String Scope: τ-effective.
Instances For
Tau.BookVI.Neural.instReprSleepRepairFunction
source instance Tau.BookVI.Neural.instReprSleepRepairFunction :Repr SleepRepairFunction
Equations
- Tau.BookVI.Neural.instReprSleepRepairFunction = { reprPrec := Tau.BookVI.Neural.instReprSleepRepairFunction.repr }
Tau.BookVI.Neural.instReprSleepRepairFunction.repr
source def Tau.BookVI.Neural.instReprSleepRepairFunction.repr :SleepRepairFunction → ℕ → Std.Format
Equations
- One or more equations did not get rendered due to their size. Instances For
Tau.BookVI.Neural.sleep_repair
source def Tau.BookVI.Neural.sleep_repair :SleepRepairFunction
Equations
- Tau.BookVI.Neural.sleep_repair = { } Instances For
Tau.BookVI.Neural.SleepConsolidatesLevels12
source structure Tau.BookVI.Neural.SleepConsolidatesLevels12 :Type
[VI.T53] Sleep Consolidates Levels 1–2 Defects. The sleep lobe of the neural temporal lemniscate (VI.P19) implements defect consolidation specifically at Levels 1 and 2 of the NeuralDefectTower (VI.D88). Proof: (1) NREM/SWS activates glymphatic clearance, which removes Level 1 molecular debris (amyloid-β, tau oligomers, metabolic waste). (2) REM activates synaptic homeostasis (Tononi–Cirelli downscaling), which maintains Level 2 synaptic integrity by pruning overfit connections. (3) Levels 3–4 operate on timescales (years–decades) that individual sleep cycles cannot address: circuit and network degradation accumulate irreversibly under the repair budget constraint (VI.D45). Scope: τ-effective.
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nrem_level_1 : Bool NREM → Level 1 glymphatic repair.
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rem_level_2 : Bool REM → Level 2 synaptic homeostasis.
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levels_3_4_excluded : Bool Levels 3–4 not addressed by sleep.
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scope : String Scope: τ-effective.
Instances For
Tau.BookVI.Neural.instReprSleepConsolidatesLevels12
source instance Tau.BookVI.Neural.instReprSleepConsolidatesLevels12 :Repr SleepConsolidatesLevels12
Equations
- Tau.BookVI.Neural.instReprSleepConsolidatesLevels12 = { reprPrec := Tau.BookVI.Neural.instReprSleepConsolidatesLevels12.repr }
Tau.BookVI.Neural.instReprSleepConsolidatesLevels12.repr
source def Tau.BookVI.Neural.instReprSleepConsolidatesLevels12.repr :SleepConsolidatesLevels12 → ℕ → Std.Format
Equations
- One or more equations did not get rendered due to their size. Instances For
Tau.BookVI.Neural.sleep_consol
source def Tau.BookVI.Neural.sleep_consol :SleepConsolidatesLevels12
Equations
- Tau.BookVI.Neural.sleep_consol = { } Instances For
Tau.BookVI.Neural.sleep_consolidates_levels_1_2
source theorem Tau.BookVI.Neural.sleep_consolidates_levels_1_2 :sleep_consol.nrem_level_1 = true ∧ sleep_consol.rem_level_2 = true ∧ sleep_consol.levels_3_4_excluded = true
Tau.BookVI.Neural.SleepDeprivationAccelerates
source structure Tau.BookVI.Neural.SleepDeprivationAccelerates :Type
[VI.P24] Sleep Deprivation Accelerates Defect Threshold Crossing. Chronic sleep deprivation skips Level 1–2 repair cycles (VI.D90), accelerating repair budget exhaustion (VI.D45) at these levels. Consequence: the Level 1 defect trajectory crosses the cascade threshold earlier, triggering accelerated Level 2 degradation via inter-level cascade (VI.T52), consistent with epidemiological evidence linking sleep deprivation to increased Alzheimer’s risk. Scope: τ-effective (structural budget argument; quantitative prediction would require empirical rates).
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budget_exhaustion_accelerated : Bool Skipped repair cycles → faster budget exhaustion.
-
level_1_earlier : Bool Level 1 threshold crossed earlier.
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cascade_earlier : Bool Cascade to Level 2 triggered earlier.
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alzheimers_consistent : Bool Consistent with Alzheimer’s epidemiology.
-
scope : String Scope: τ-effective.
Instances For
Tau.BookVI.Neural.instReprSleepDeprivationAccelerates
source instance Tau.BookVI.Neural.instReprSleepDeprivationAccelerates :Repr SleepDeprivationAccelerates
Equations
- Tau.BookVI.Neural.instReprSleepDeprivationAccelerates = { reprPrec := Tau.BookVI.Neural.instReprSleepDeprivationAccelerates.repr }
Tau.BookVI.Neural.instReprSleepDeprivationAccelerates.repr
source def Tau.BookVI.Neural.instReprSleepDeprivationAccelerates.repr :SleepDeprivationAccelerates → ℕ → Std.Format
Equations
- One or more equations did not get rendered due to their size. Instances For
Tau.BookVI.Neural.sleep_dep
source def Tau.BookVI.Neural.sleep_dep :SleepDeprivationAccelerates
Equations
- Tau.BookVI.Neural.sleep_dep = { } Instances For
Tau.BookVI.Neural.sleep_deprivation_accelerates
source theorem Tau.BookVI.Neural.sleep_deprivation_accelerates :sleep_dep.budget_exhaustion_accelerated = true ∧ sleep_dep.level_1_earlier = true ∧ sleep_dep.cascade_earlier = true ∧ sleep_dep.alzheimers_consistent = true
Tau.BookVI.Neural.NeuralHayflickBound
source structure Tau.BookVI.Neural.NeuralHayflickBound :Type
[VI.D91] Neural Hayflick Bound: maximum cognitive lifespan at each defect level, derived from finite repair budget (VI.P16) applied to the NeuralDefectTower (VI.D88). H_i = R_max(i) / r_i, where R_max(i) is the repair budget allocated to Level i and r_i is the defect accumulation rate. Overall cognitive Hayflick bound: H_neural = min(H₁,H₂,H₃,H₄). Connects to Book V: the geometric decay rate (1−ι_τ)^n (V.T62) governs the baseline defect accumulation at each level. Scope: τ-effective.
-
level_count : ℕ Number of levels with individual bounds.
-
count_eq : self.level_count = 4 4 individual bounds.
-
finite_per_level : Bool Each level has a finite Hayflick bound H_i.
-
overall_is_min : Bool Overall bound is min of level bounds.
-
connects_to_book_v : Bool Connects to Book V defect exhaustion (V.T62).
-
scope : String Scope: τ-effective.
Instances For
Tau.BookVI.Neural.instReprNeuralHayflickBound.repr
source def Tau.BookVI.Neural.instReprNeuralHayflickBound.repr :NeuralHayflickBound → ℕ → Std.Format
Equations
- One or more equations did not get rendered due to their size. Instances For
Tau.BookVI.Neural.instReprNeuralHayflickBound
source instance Tau.BookVI.Neural.instReprNeuralHayflickBound :Repr NeuralHayflickBound
Equations
- Tau.BookVI.Neural.instReprNeuralHayflickBound = { reprPrec := Tau.BookVI.Neural.instReprNeuralHayflickBound.repr }
Tau.BookVI.Neural.neural_hayflick
source def Tau.BookVI.Neural.neural_hayflick :NeuralHayflickBound
Equations
- Tau.BookVI.Neural.neural_hayflick = { level_count := 4, count_eq := Tau.BookVI.Neural.neural_tower._proof_1 } Instances For
Tau.BookVI.Neural.NeurodegenerationHayflickCrossing
source structure Tau.BookVI.Neural.NeurodegenerationHayflickCrossing :Type
[VI.T54] Neurodegeneration = Hayflick Crossing. A neurodegenerative disease occurs when a specific level’s Hayflick bound H_i is exhausted before the organismal Hayflick limit: the repair budget at Level i is depleted, defects accumulate past the cascade threshold, and cognitive function degrades irreversibly. Alzheimer’s: H₁ exhausted first (molecular repair depleted). Parkinson’s: H₃ exhausted first (circuit repair depleted). Normal aging: all H_i > organismal limit (no level crosses first). The neural Hayflick bound is a sector-specific instance of the universal defect exhaustion (V.T62/VI.P16), with (1−ι_τ)^n governing the baseline. Scope: τ-effective.
-
disease_is_level_crossing : Bool Disease = specific level Hayflick bound exhausted.
-
alzheimers_h1 : Bool Alzheimer’s = H₁ first.
-
parkinsons_h3 : Bool Parkinson’s = H₃ first.
-
normal_aging_safe : Bool Normal aging: no H_i crossed before organismal limit.
-
specializes_universal : Bool Sector-specific instance of V.T62/VI.P16.
-
scope : String Scope: τ-effective.
Instances For
Tau.BookVI.Neural.instReprNeurodegenerationHayflickCrossing
source instance Tau.BookVI.Neural.instReprNeurodegenerationHayflickCrossing :Repr NeurodegenerationHayflickCrossing
Equations
- Tau.BookVI.Neural.instReprNeurodegenerationHayflickCrossing = { reprPrec := Tau.BookVI.Neural.instReprNeurodegenerationHayflickCrossing.repr }
Tau.BookVI.Neural.instReprNeurodegenerationHayflickCrossing.repr
source def Tau.BookVI.Neural.instReprNeurodegenerationHayflickCrossing.repr :NeurodegenerationHayflickCrossing → ℕ → Std.Format
Equations
- One or more equations did not get rendered due to their size. Instances For
Tau.BookVI.Neural.neuro_crossing
source def Tau.BookVI.Neural.neuro_crossing :NeurodegenerationHayflickCrossing
Equations
- Tau.BookVI.Neural.neuro_crossing = { } Instances For
Tau.BookVI.Neural.neurodegeneration_is_hayflick_crossing
source theorem Tau.BookVI.Neural.neurodegeneration_is_hayflick_crossing :neuro_crossing.disease_is_level_crossing = true ∧ neuro_crossing.alzheimers_h1 = true ∧ neuro_crossing.parkinsons_h3 = true ∧ neuro_crossing.normal_aging_safe = true ∧ neuro_crossing.specializes_universal = true