TauLib · API Book VI

TauLib.BookVI.Consumer.Neural

TauLib.BookVI.Consumer.Neural

Neural systems as τ³ computers, and sleep as the temporal lemniscate’s second lobe.

Registry Cross-References

  • [VI.D52] Neural Architecture as τ³ Computer — NeuralArchitecture

  • [VI.P19] Sleep as Temporal Lemniscate Second Lobe — sleep_two_lobes

Cross-Book Authority

  • Book II, Part II: τ³ = τ¹ ×_f T² fibration (neural architecture mirrors τ³ structure)

  • Book VI, Part 2: Temporal lemniscate L_T (persistence sector, circadian rhythm)

  • Book III, Part I: P vs NP force (cognitive optimization)

Ground Truth Sources

  • Book VI Chapter 40 (2nd Edition): Neural Systems

  • Book VI Chapter 41 (2nd Edition): Learning and Sleep

Wave R7-E: Neural Defect Accumulation (2026-03-08)

  • [VI.D87] Neural Defect Level — NeuralDefectLevel

  • [VI.D88] Neural Defect Tower — NeuralDefectTower

  • [VI.D89] Neurodegenerative Disease Mapping — NeurodegenerativeMapping

  • [VI.T52] Inter-Level Cascade — inter_level_cascade

  • [VI.P23] Neural Defect Monotonicity — neural_defect_monotone

  • [VI.D90] Sleep Repair Function — SleepRepairFunction

  • [VI.T53] Sleep Consolidates Levels 1–2 — sleep_consolidates_levels_1_2

  • [VI.P24] Sleep Deprivation Accelerates — sleep_deprivation_accelerates

  • [VI.D91] Neural Hayflick Bound — NeuralHayflickBound

  • [VI.T54] Neurodegeneration = Hayflick Crossing — neurodegeneration_is_hayflick_crossing


Tau.BookVI.Neural.NeuralArchitecture

source structure Tau.BookVI.Neural.NeuralArchitecture :Type

[VI.D52] Neural Architecture as τ³ Computer. Three node types: sensory (input), inter (processing), motor (output). Weighted directed edges. The architecture mirrors the τ³ fibration (Book II, Part II): base τ¹ = temporal sequencing, fiber T² = parallel feature processing.

  • node_types : ℕ Number of fundamental node types.

  • types_eq : self.node_types = 3 Exactly 3 types: sensory, inter, motor.

  • weighted_edges : Bool Edges carry weights.

  • directed : Bool Network is directed (sensory → inter → motor).

  • tau3_computer : Bool Architecture mirrors τ³ structure.

Instances For


Tau.BookVI.Neural.instReprNeuralArchitecture

source instance Tau.BookVI.Neural.instReprNeuralArchitecture :Repr NeuralArchitecture

Equations

  • Tau.BookVI.Neural.instReprNeuralArchitecture = { reprPrec := Tau.BookVI.Neural.instReprNeuralArchitecture.repr }

Tau.BookVI.Neural.instReprNeuralArchitecture.repr

source def Tau.BookVI.Neural.instReprNeuralArchitecture.repr :NeuralArchitecture → ℕ → Std.Format

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Tau.BookVI.Neural.neural_arch

source def Tau.BookVI.Neural.neural_arch :NeuralArchitecture

Equations

  • Tau.BookVI.Neural.neural_arch = { node_types := 3, types_eq := Tau.BookVI.Neural.neural_arch._proof_1 } Instances For

Tau.BookVI.Neural.neural_is_tau3_computer

source theorem Tau.BookVI.Neural.neural_is_tau3_computer :neural_arch.node_types = 3 ∧ neural_arch.directed = true ∧ neural_arch.tau3_computer = true


Tau.BookVI.Neural.SleepLemniscate

source structure Tau.BookVI.Neural.SleepLemniscate :Type

[VI.P19] Sleep as Temporal Lemniscate Second Lobe. The temporal lemniscate L_T = S¹ ∨ S¹ (Book VI, Part 2) has two lobes: wakefulness (active processing) and sleep (consolidation/pruning). Circadian rhythm is the orbit traversing both lobes.

  • lobe_count : ℕ Number of lemniscate lobes.

  • count_eq : self.lobe_count = 2 Exactly 2 lobes.

  • wake_lobe : Bool Lobe 1: wakefulness.

  • sleep_lobe : Bool Lobe 2: sleep.

  • circadian_link : Bool Linked to circadian rhythm (Part 2).

Instances For


Tau.BookVI.Neural.instReprSleepLemniscate.repr

source def Tau.BookVI.Neural.instReprSleepLemniscate.repr :SleepLemniscate → ℕ → Std.Format

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Tau.BookVI.Neural.instReprSleepLemniscate

source instance Tau.BookVI.Neural.instReprSleepLemniscate :Repr SleepLemniscate

Equations

  • Tau.BookVI.Neural.instReprSleepLemniscate = { reprPrec := Tau.BookVI.Neural.instReprSleepLemniscate.repr }

Tau.BookVI.Neural.sleep_lemn

source def Tau.BookVI.Neural.sleep_lemn :SleepLemniscate

Equations

  • Tau.BookVI.Neural.sleep_lemn = { lobe_count := 2, count_eq := Tau.BookVI.Neural.sleep_lemn._proof_1 } Instances For

Tau.BookVI.Neural.sleep_two_lobes

source theorem Tau.BookVI.Neural.sleep_two_lobes :sleep_lemn.lobe_count = 2 ∧ sleep_lemn.wake_lobe = true ∧ sleep_lemn.sleep_lobe = true ∧ sleep_lemn.circadian_link = true


Tau.BookVI.Neural.NeuralDefectLevel

source inductive Tau.BookVI.Neural.NeuralDefectLevel :Type

[VI.D87] Neural Defect Level: 4 hierarchical levels of neural defect accumulation, specializing VI.D43 (AgingDefect) to the neural architecture (VI.D52). Level 1 — Molecular: protein misfolding, aggregation (amyloid-β, α-synuclein, tau tangles). Defect = deviation from native fold. Level 2 — Synaptic: synapse loss, neurotransmitter depletion, receptor downregulation. Defect = edge degradation in τ³-computer. Level 3 — Circuit: dopaminergic/serotonergic/cholinergic pathway degradation. Defect = subgraph integrity loss in τ³-computer. Level 4 — Network: large-scale connectivity loss, white matter degeneration. Defect = global topology disruption in τ³-computer. Scope: τ-effective.

  • molecular : NeuralDefectLevel
  • synaptic : NeuralDefectLevel
  • circuit : NeuralDefectLevel
  • network : NeuralDefectLevel Instances For

Tau.BookVI.Neural.instReprNeuralDefectLevel.repr

source def Tau.BookVI.Neural.instReprNeuralDefectLevel.repr :NeuralDefectLevel → ℕ → Std.Format

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Tau.BookVI.Neural.instReprNeuralDefectLevel

source instance Tau.BookVI.Neural.instReprNeuralDefectLevel :Repr NeuralDefectLevel

Equations

  • Tau.BookVI.Neural.instReprNeuralDefectLevel = { reprPrec := Tau.BookVI.Neural.instReprNeuralDefectLevel.repr }

Tau.BookVI.Neural.instBEqNeuralDefectLevel.beq

source def Tau.BookVI.Neural.instBEqNeuralDefectLevel.beq :NeuralDefectLevel → NeuralDefectLevel → Bool

Equations

  • Tau.BookVI.Neural.instBEqNeuralDefectLevel.beq x✝ y✝ = (x✝.ctorIdx == y✝.ctorIdx) Instances For

Tau.BookVI.Neural.instBEqNeuralDefectLevel

source instance Tau.BookVI.Neural.instBEqNeuralDefectLevel :BEq NeuralDefectLevel

Equations

  • Tau.BookVI.Neural.instBEqNeuralDefectLevel = { beq := Tau.BookVI.Neural.instBEqNeuralDefectLevel.beq }

Tau.BookVI.Neural.NeuralDefectTower

source structure Tau.BookVI.Neural.NeuralDefectTower :Type

[VI.D88] Neural Defect Tower: multi-level defect accumulation specialized to the neural architecture (VI.D52). Each level i has a defect functional Δᵢ(n) that is monotonically increasing with refinement step n (specialization of VI.D43). Levels cascade: when Level i defect exceeds a threshold, Level i+1 defect accumulation accelerates. Scope: τ-effective.

  • level_count : ℕ Number of hierarchical levels.

  • count_eq : self.level_count = 4 Exactly 4 levels.

  • monotone_per_level : Bool Each level’s defect is monotonically increasing (VI.D43).

  • inter_level_cascade : Bool Levels cascade: Level i overflow accelerates Level i+1.

  • specializes_aging_defect : Bool Specialization of aging defect (VI.D43).

  • scope : String Scope: τ-effective.

Instances For


Tau.BookVI.Neural.instReprNeuralDefectTower.repr

source def Tau.BookVI.Neural.instReprNeuralDefectTower.repr :NeuralDefectTower → ℕ → Std.Format

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Tau.BookVI.Neural.instReprNeuralDefectTower

source instance Tau.BookVI.Neural.instReprNeuralDefectTower :Repr NeuralDefectTower

Equations

  • Tau.BookVI.Neural.instReprNeuralDefectTower = { reprPrec := Tau.BookVI.Neural.instReprNeuralDefectTower.repr }

Tau.BookVI.Neural.neural_tower

source def Tau.BookVI.Neural.neural_tower :NeuralDefectTower

Equations

  • Tau.BookVI.Neural.neural_tower = { level_count := 4, count_eq := Tau.BookVI.Neural.neural_tower._proof_1 } Instances For

Tau.BookVI.Neural.NeurodegenerativeMapping

source structure Tau.BookVI.Neural.NeurodegenerativeMapping :Type

[VI.D89] Neurodegenerative Disease Mapping: each major neurodegenerative disease is characterized by a dominant defect level at which repair budget exhaustion occurs first. Alzheimer’s: Level 1 dominant (amyloid/tau aggregation). Parkinson’s: Level 3 dominant (dopaminergic circuit loss). ALS: Level 3 dominant (motor neuron circuit failure). Huntington’s: Level 1 dominant (polyQ aggregation). Normal aging: all levels degrade but none crosses threshold before organismal Hayflick limit. Scope: τ-effective (structural classification; protein names appear in documentation only, not in formal conditions).

  • alzheimers_level : NeuralDefectLevel Alzheimer’s: molecular level dominant.

  • parkinsons_level : NeuralDefectLevel Parkinson’s: circuit level dominant.

  • als_level : NeuralDefectLevel ALS: circuit level dominant.

  • huntingtons_level : NeuralDefectLevel Huntington’s: molecular level dominant.

  • scope : String Scope: τ-effective.

Instances For


Tau.BookVI.Neural.instReprNeurodegenerativeMapping

source instance Tau.BookVI.Neural.instReprNeurodegenerativeMapping :Repr NeurodegenerativeMapping

Equations

  • Tau.BookVI.Neural.instReprNeurodegenerativeMapping = { reprPrec := Tau.BookVI.Neural.instReprNeurodegenerativeMapping.repr }

Tau.BookVI.Neural.instReprNeurodegenerativeMapping.repr

source def Tau.BookVI.Neural.instReprNeurodegenerativeMapping.repr :NeurodegenerativeMapping → ℕ → Std.Format

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Tau.BookVI.Neural.disease_map

source def Tau.BookVI.Neural.disease_map :NeurodegenerativeMapping

Equations

  • Tau.BookVI.Neural.disease_map = { } Instances For

Tau.BookVI.Neural.InterLevelCascade

source structure Tau.BookVI.Neural.InterLevelCascade :Type

[VI.T52] Inter-Level Cascade Theorem. Level i defect accumulation beyond threshold triggers accelerated defect accumulation at Level i+1 (upward cascade). Proof: (1) Molecular aggregates (Level 1) impair synaptic transmission (Level 2) by disrupting vesicle trafficking and receptor function. (2) Synaptic loss (Level 2) degrades circuit integrity (Level 3) by removing edges from the τ³-computer subgraph. (3) Circuit degradation (Level 3) fragments the global network (Level 4) by disconnecting integrative pathways. Each transition is monotone: more Level i defect → more Level i+1 defect. The cascade is unidirectional (upward only). Scope: τ-effective.

  • molecular_to_synaptic : Bool Level 1 → Level 2 cascade.

  • synaptic_to_circuit : Bool Level 2 → Level 3 cascade.

  • circuit_to_network : Bool Level 3 → Level 4 cascade.

  • upward_only : Bool Cascade is unidirectional (upward).

  • scope : String Scope: τ-effective.

Instances For


Tau.BookVI.Neural.instReprInterLevelCascade.repr

source def Tau.BookVI.Neural.instReprInterLevelCascade.repr :InterLevelCascade → ℕ → Std.Format

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Tau.BookVI.Neural.instReprInterLevelCascade

source instance Tau.BookVI.Neural.instReprInterLevelCascade :Repr InterLevelCascade

Equations

  • Tau.BookVI.Neural.instReprInterLevelCascade = { reprPrec := Tau.BookVI.Neural.instReprInterLevelCascade.repr }

Tau.BookVI.Neural.cascade

source def Tau.BookVI.Neural.cascade :InterLevelCascade

Equations

  • Tau.BookVI.Neural.cascade = { } Instances For

Tau.BookVI.Neural.inter_level_cascade

source theorem Tau.BookVI.Neural.inter_level_cascade :cascade.molecular_to_synaptic = true ∧ cascade.synaptic_to_circuit = true ∧ cascade.circuit_to_network = true ∧ cascade.upward_only = true


Tau.BookVI.Neural.NeuralDefectMonotone

source structure Tau.BookVI.Neural.NeuralDefectMonotone :Type

[VI.P23] Neural Defect Monotonicity. At each level i of the NeuralDefectTower, the defect functional Δᵢ(n) is monotonically non-decreasing in the refinement step n. This is a specialization of VI.D43 (AgingDefect: Δ(n) monotonically increasing) to the 4-level neural decomposition: the total neural defect Δ_neural(n) = Σᵢ Δᵢ(n) inherits monotonicity from each component, and each component inherits it from VI.D43 restricted to the neural subsystem. Scope: τ-effective.

  • per_level_monotone : Bool Each level’s defect is monotone non-decreasing.

  • total_monotone : Bool Total neural defect inherits monotonicity.

  • specializes_d43 : Bool Specializes VI.D43.

  • scope : String Scope: τ-effective.

Instances For


Tau.BookVI.Neural.instReprNeuralDefectMonotone.repr

source def Tau.BookVI.Neural.instReprNeuralDefectMonotone.repr :NeuralDefectMonotone → ℕ → Std.Format

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Tau.BookVI.Neural.instReprNeuralDefectMonotone

source instance Tau.BookVI.Neural.instReprNeuralDefectMonotone :Repr NeuralDefectMonotone

Equations

  • Tau.BookVI.Neural.instReprNeuralDefectMonotone = { reprPrec := Tau.BookVI.Neural.instReprNeuralDefectMonotone.repr }

Tau.BookVI.Neural.neural_mono

source def Tau.BookVI.Neural.neural_mono :NeuralDefectMonotone

Equations

  • Tau.BookVI.Neural.neural_mono = { } Instances For

Tau.BookVI.Neural.neural_defect_monotone

source theorem Tau.BookVI.Neural.neural_defect_monotone :neural_mono.per_level_monotone = true ∧ neural_mono.total_monotone = true ∧ neural_mono.specializes_d43 = true


Tau.BookVI.Neural.SleepRepairFunction

source structure Tau.BookVI.Neural.SleepRepairFunction :Type

[VI.D90] Sleep Repair Function: dual-lobe repair at specific NeuralDefectTower levels, using the sleep lemniscate (VI.P19). NREM/SWS (S¹_sleep Lobe 1): Level 1 repair — glymphatic clearance removes molecular debris (amyloid-β, metabolic waste). REM (S¹_sleep Lobe 2): Level 2 repair — synaptic homeostasis, memory consolidation, pruning of weak connections. Levels 3–4 are NOT repaired by sleep: circuit and network degradation are irreversible once established (repair budget does not cover these levels at the rate they accumulate). Scope: τ-effective.

  • nrem_repairs_molecular : Bool NREM repairs Level 1 (molecular/glymphatic).

  • rem_repairs_synaptic : Bool REM repairs Level 2 (synaptic homeostasis).

  • no_circuit_repair : Bool Level 3 not repaired by sleep.

  • no_network_repair : Bool Level 4 not repaired by sleep.

  • consumes_repair_budget : Bool Each sleep cycle consumes repair budget (VI.D45).

  • scope : String Scope: τ-effective.

Instances For


Tau.BookVI.Neural.instReprSleepRepairFunction

source instance Tau.BookVI.Neural.instReprSleepRepairFunction :Repr SleepRepairFunction

Equations

  • Tau.BookVI.Neural.instReprSleepRepairFunction = { reprPrec := Tau.BookVI.Neural.instReprSleepRepairFunction.repr }

Tau.BookVI.Neural.instReprSleepRepairFunction.repr

source def Tau.BookVI.Neural.instReprSleepRepairFunction.repr :SleepRepairFunction → ℕ → Std.Format

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  • One or more equations did not get rendered due to their size. Instances For

Tau.BookVI.Neural.sleep_repair

source def Tau.BookVI.Neural.sleep_repair :SleepRepairFunction

Equations

  • Tau.BookVI.Neural.sleep_repair = { } Instances For

Tau.BookVI.Neural.SleepConsolidatesLevels12

source structure Tau.BookVI.Neural.SleepConsolidatesLevels12 :Type

[VI.T53] Sleep Consolidates Levels 1–2 Defects. The sleep lobe of the neural temporal lemniscate (VI.P19) implements defect consolidation specifically at Levels 1 and 2 of the NeuralDefectTower (VI.D88). Proof: (1) NREM/SWS activates glymphatic clearance, which removes Level 1 molecular debris (amyloid-β, tau oligomers, metabolic waste). (2) REM activates synaptic homeostasis (Tononi–Cirelli downscaling), which maintains Level 2 synaptic integrity by pruning overfit connections. (3) Levels 3–4 operate on timescales (years–decades) that individual sleep cycles cannot address: circuit and network degradation accumulate irreversibly under the repair budget constraint (VI.D45). Scope: τ-effective.

  • nrem_level_1 : Bool NREM → Level 1 glymphatic repair.

  • rem_level_2 : Bool REM → Level 2 synaptic homeostasis.

  • levels_3_4_excluded : Bool Levels 3–4 not addressed by sleep.

  • scope : String Scope: τ-effective.

Instances For


Tau.BookVI.Neural.instReprSleepConsolidatesLevels12

source instance Tau.BookVI.Neural.instReprSleepConsolidatesLevels12 :Repr SleepConsolidatesLevels12

Equations

  • Tau.BookVI.Neural.instReprSleepConsolidatesLevels12 = { reprPrec := Tau.BookVI.Neural.instReprSleepConsolidatesLevels12.repr }

Tau.BookVI.Neural.instReprSleepConsolidatesLevels12.repr

source def Tau.BookVI.Neural.instReprSleepConsolidatesLevels12.repr :SleepConsolidatesLevels12 → ℕ → Std.Format

Equations

  • One or more equations did not get rendered due to their size. Instances For

Tau.BookVI.Neural.sleep_consol

source def Tau.BookVI.Neural.sleep_consol :SleepConsolidatesLevels12

Equations

  • Tau.BookVI.Neural.sleep_consol = { } Instances For

Tau.BookVI.Neural.sleep_consolidates_levels_1_2

source theorem Tau.BookVI.Neural.sleep_consolidates_levels_1_2 :sleep_consol.nrem_level_1 = true ∧ sleep_consol.rem_level_2 = true ∧ sleep_consol.levels_3_4_excluded = true


Tau.BookVI.Neural.SleepDeprivationAccelerates

source structure Tau.BookVI.Neural.SleepDeprivationAccelerates :Type

[VI.P24] Sleep Deprivation Accelerates Defect Threshold Crossing. Chronic sleep deprivation skips Level 1–2 repair cycles (VI.D90), accelerating repair budget exhaustion (VI.D45) at these levels. Consequence: the Level 1 defect trajectory crosses the cascade threshold earlier, triggering accelerated Level 2 degradation via inter-level cascade (VI.T52), consistent with epidemiological evidence linking sleep deprivation to increased Alzheimer’s risk. Scope: τ-effective (structural budget argument; quantitative prediction would require empirical rates).

  • budget_exhaustion_accelerated : Bool Skipped repair cycles → faster budget exhaustion.

  • level_1_earlier : Bool Level 1 threshold crossed earlier.

  • cascade_earlier : Bool Cascade to Level 2 triggered earlier.

  • alzheimers_consistent : Bool Consistent with Alzheimer’s epidemiology.

  • scope : String Scope: τ-effective.

Instances For


Tau.BookVI.Neural.instReprSleepDeprivationAccelerates

source instance Tau.BookVI.Neural.instReprSleepDeprivationAccelerates :Repr SleepDeprivationAccelerates

Equations

  • Tau.BookVI.Neural.instReprSleepDeprivationAccelerates = { reprPrec := Tau.BookVI.Neural.instReprSleepDeprivationAccelerates.repr }

Tau.BookVI.Neural.instReprSleepDeprivationAccelerates.repr

source def Tau.BookVI.Neural.instReprSleepDeprivationAccelerates.repr :SleepDeprivationAccelerates → ℕ → Std.Format

Equations

  • One or more equations did not get rendered due to their size. Instances For

Tau.BookVI.Neural.sleep_dep

source def Tau.BookVI.Neural.sleep_dep :SleepDeprivationAccelerates

Equations

  • Tau.BookVI.Neural.sleep_dep = { } Instances For

Tau.BookVI.Neural.sleep_deprivation_accelerates

source theorem Tau.BookVI.Neural.sleep_deprivation_accelerates :sleep_dep.budget_exhaustion_accelerated = true ∧ sleep_dep.level_1_earlier = true ∧ sleep_dep.cascade_earlier = true ∧ sleep_dep.alzheimers_consistent = true


Tau.BookVI.Neural.NeuralHayflickBound

source structure Tau.BookVI.Neural.NeuralHayflickBound :Type

[VI.D91] Neural Hayflick Bound: maximum cognitive lifespan at each defect level, derived from finite repair budget (VI.P16) applied to the NeuralDefectTower (VI.D88). H_i = R_max(i) / r_i, where R_max(i) is the repair budget allocated to Level i and r_i is the defect accumulation rate. Overall cognitive Hayflick bound: H_neural = min(H₁,H₂,H₃,H₄). Connects to Book V: the geometric decay rate (1−ι_τ)^n (V.T62) governs the baseline defect accumulation at each level. Scope: τ-effective.

  • level_count : ℕ Number of levels with individual bounds.

  • count_eq : self.level_count = 4 4 individual bounds.

  • finite_per_level : Bool Each level has a finite Hayflick bound H_i.

  • overall_is_min : Bool Overall bound is min of level bounds.

  • connects_to_book_v : Bool Connects to Book V defect exhaustion (V.T62).

  • scope : String Scope: τ-effective.

Instances For


Tau.BookVI.Neural.instReprNeuralHayflickBound.repr

source def Tau.BookVI.Neural.instReprNeuralHayflickBound.repr :NeuralHayflickBound → ℕ → Std.Format

Equations

  • One or more equations did not get rendered due to their size. Instances For

Tau.BookVI.Neural.instReprNeuralHayflickBound

source instance Tau.BookVI.Neural.instReprNeuralHayflickBound :Repr NeuralHayflickBound

Equations

  • Tau.BookVI.Neural.instReprNeuralHayflickBound = { reprPrec := Tau.BookVI.Neural.instReprNeuralHayflickBound.repr }

Tau.BookVI.Neural.neural_hayflick

source def Tau.BookVI.Neural.neural_hayflick :NeuralHayflickBound

Equations

  • Tau.BookVI.Neural.neural_hayflick = { level_count := 4, count_eq := Tau.BookVI.Neural.neural_tower._proof_1 } Instances For

Tau.BookVI.Neural.NeurodegenerationHayflickCrossing

source structure Tau.BookVI.Neural.NeurodegenerationHayflickCrossing :Type

[VI.T54] Neurodegeneration = Hayflick Crossing. A neurodegenerative disease occurs when a specific level’s Hayflick bound H_i is exhausted before the organismal Hayflick limit: the repair budget at Level i is depleted, defects accumulate past the cascade threshold, and cognitive function degrades irreversibly. Alzheimer’s: H₁ exhausted first (molecular repair depleted). Parkinson’s: H₃ exhausted first (circuit repair depleted). Normal aging: all H_i > organismal limit (no level crosses first). The neural Hayflick bound is a sector-specific instance of the universal defect exhaustion (V.T62/VI.P16), with (1−ι_τ)^n governing the baseline. Scope: τ-effective.

  • disease_is_level_crossing : Bool Disease = specific level Hayflick bound exhausted.

  • alzheimers_h1 : Bool Alzheimer’s = H₁ first.

  • parkinsons_h3 : Bool Parkinson’s = H₃ first.

  • normal_aging_safe : Bool Normal aging: no H_i crossed before organismal limit.

  • specializes_universal : Bool Sector-specific instance of V.T62/VI.P16.

  • scope : String Scope: τ-effective.

Instances For


Tau.BookVI.Neural.instReprNeurodegenerationHayflickCrossing

source instance Tau.BookVI.Neural.instReprNeurodegenerationHayflickCrossing :Repr NeurodegenerationHayflickCrossing

Equations

  • Tau.BookVI.Neural.instReprNeurodegenerationHayflickCrossing = { reprPrec := Tau.BookVI.Neural.instReprNeurodegenerationHayflickCrossing.repr }

Tau.BookVI.Neural.instReprNeurodegenerationHayflickCrossing.repr

source def Tau.BookVI.Neural.instReprNeurodegenerationHayflickCrossing.repr :NeurodegenerationHayflickCrossing → ℕ → Std.Format

Equations

  • One or more equations did not get rendered due to their size. Instances For

Tau.BookVI.Neural.neuro_crossing

source def Tau.BookVI.Neural.neuro_crossing :NeurodegenerationHayflickCrossing

Equations

  • Tau.BookVI.Neural.neuro_crossing = { } Instances For

Tau.BookVI.Neural.neurodegeneration_is_hayflick_crossing

source theorem Tau.BookVI.Neural.neurodegeneration_is_hayflick_crossing :neuro_crossing.disease_is_level_crossing = true ∧ neuro_crossing.alzheimers_h1 = true ∧ neuro_crossing.parkinsons_h3 = true ∧ neuro_crossing.normal_aging_safe = true ∧ neuro_crossing.specializes_universal = true